Immediate Baby Care Airway - Clean mouth and nose Thermoregulation - Warmth APGAR Gross assessment Identification Bonding – safety against infection Medications
Fetus to Newborn: Respiratory Changes Initiation of respirations Chemical surfactant reduces surface tension 34-36wks decrease in oxygen concentration Thermal sudden chilling of moist infant Mechanical compression of fetal chest during delivery normal handling
Nursing Process for Respirations Assess for respiratory distress Plan: Maintain patent airway Interventions - Positioning infant – head lower - Suction secretions – bulb, keep near head, mouth first, avoid trauma to membranes Evaluation – rate 30-60, no distress
Fetus to Newborn: Neurological adaptation: Thermoregulation Methods of heat loss Evaporation – wet surface exposed to air Conduction – direct contact with cool objects Convection- surrounding cool air - drafts Radiation – transfer of heat to cooler objects not in direct contact with infant
Nursing Care – Cold Stress Preventing heat loss – radiant warmer Providing immediate care - dry quickly, cover head with cap, replace wet blankets Providing on going prevention - safety Restoring thermoregulation – if becoming chilled - intervene
Effects of Cold Stress Increased oxygen need Decreased surfactant production Respiratory distress Hypoglycemia Metabolic acidosis Jaundice
APGAR Heart rate – above 100 Respiratory Effort – spontaneous with cry Muscle tone – flexed with movement Reflex response – active, prompt cry Color – pink or acrocyanosis 0-3 infant needs resuscitation 4-7 Gentle stimulation – Narcan 8-10 – no action needed
Early Assessments Assess for anomalies Head – anterior fontanelle closes 12-18 mo posterior fontanelle closes 2-3 months Neck and clavicles fracture of clavicle – large infant, lump, tenderness, crepitus, decreased movement Cord Extremities flexed and resist extension assess fractures, clubfeet hips vertebral column
Measurements Weight – loss of 10% normal Length Head and chest circumference Normal VS temp 97.7-99.5F axillary apical pulse 120-160bpm respirations 30-60/min
Assessment of Cardio-respiratory Status History Airway Assess rate q 30minX2hrs symmetry breath sounds - moisture for 1-2 hrs
Assessment of Thermoregulation Check soon after birth Set warmer controls Rectal for first temp Insert only 0.5 inch Axillary route rest of time
Assessment of Hepatic Function Blood Glucose Signs of hypoglycemia jitteriness respiratory difficulties drop in temp poor sucking Tx- feed infant if glucose below 40-45 mg/dl Bilirubin physiologic jaundice peaks 2-4 days of life early onset may be pathologic
Jaundice Hemolysis of excessive erythrocytes Short red blood cell life Liver immaturity Lack of intestinal flora Delayed feeding Trauma resulting in bruising or cephalhematoma Cold stress or asphyxia
Assessment of Neuro System Reflexes Babinski Grasp Moro Rooting Stepping Sucking Tonic neck reflex “fencing” Cry Infant response to soothing
Assessment of Gastrointestinal System Mouth Suck Abdomen Initial feeding Stools meconium – within 12-48 hours of birth dark greenish black breastfed – soft, seedy, mustard yellow formula-fed – solid, pale yellow
Assessment of Genitourinary System Umbilical cord vessels Urine – within 24 hours of birth Voiding – 6 to 10 times a day after 2 days Genitalia female – edema normal, majora covers minora, pseudomenstruation male – pendulous scrotum, descended testes by 36 wks gest., placement of meatus
Assessment of Integumentary System Vernix – white covering Lanugo – fine hair Milia Erythema toxicum – red blotchy with white Birthmarks Mongolian spots – sacral area Telangiectatic nevus “stork Bite” - blanches Nevus flammeus “port wine stain” - no blanching Nevus vasculosus “strawberry hemangioma” usually on head, disappears by school age
Fetus to Newborn: Psychosocial adaptation Periods of Reactivity active – 30-60 min sleep – 2-4 hours alert – 4-6 hours Behavioral States quiet sleep active sleep drowsy state quiet alert – best for bonding active alert crying state
Gestational Age Assessment Assessment tool – Dubowitz, Ballard Weeks from conception to birth Used to identify high risk infants Neuromuscular characteristics Posture – more flexion Square window – more pliable Arm recoil - active Popliteal angle - less Scarf Sign – less crossing Heel to ear – most resistance
Gestational Age Assessment Physical characteristics Skin- deep cracking, no vessels seen, post-leathery Lanugo – less as age Plantar creases – more with age Breasts – larger areola Eyes and Ears – stiff with instant recoil Genitals – deep rugae, pendulous, covers minora Gestational Age & Size – may not correspond small SGA <10% for weight large LGA >90% for weight appropriate AGA between 10-90%
Ongoing Assessment and Care Bathing Cord care Cleansing diaper area Assisting with feedings Protecting infant identifying infant preventing infant abduction – alert to unusual preventing infection Review beige cue cards in center of book for teach
One method of swaddling a baby.
Circumcision Most common neonatal surgical procedure Reasons for choosing Reasons for rejecting – hypospadias, epispadias Pain relief Methods Nursing care
Other Concerns Immunizations Hepatitis B – begin vaccine at birth Screening tests Hearing Phenylketonuria – by law
Further Assessments Complications r/t poorly functioning placenta hypoglycemia hypothermia respiratory problems
Shoulder Dystocia Risk factors diabetes; macrosomic infant obesity prolonged second stage previous shoulder dystocia Morbidity- fracture of clavicle or humerus, brachial plexus injury Management – generous episiotomy
High Risk Infants Preterm – before 38 weeks gestation IUGR – full term but failed to grow normally SGA - LGA Infants of Diabetic mothers Post mature babies Drug exposed
Preterm infants Survive - Weight 1250 g -1500 g – 85-90% 500-600g at birth 20% survive Ethical questions Characteristics – frail, weak, limp, skin translucent, abundant vernix & lanugo Behavior – easily exhausted, from noise and routine activities, feeble cry
Nursing Care of Preterm Infants Inadequate respirations Inadequate thermoregulation Fluid and electrolyte imbalance – dehydration sunken fontanels <1ml/kg/hr or over hydration bulging, edema and urine output >3ml/kg/hr Signs of pain – high-pitched cry, >VS Signs of over stimulation - >P, >RR, stiff extended extremities, turning face away Nutrition – signs of readiness to nipple resp <60/m, rooting, sucking, gag reflex
Complications of Preterm Infants Respiratory Distress Syndrome -RDS Periventricular-Intraventricular Hemorrhage 30% infants <32 wk gest or <1500 g Retrolenthal fibroplasia – visual impairment or blindness from O2 & ventilator Necrotizing Enterocolitis (NEC) – distention, increased residual, Tx - rest bowel
Respiratory Distress Syndrome RDS also know as “hyaline membrane disease” Cause – besides preemie, C/S, diabetic mothers, birth asphyxia – interfere with surfactant S & S tachypnea - over 60/min retractions- sternal or intercostal nasal flaring cyanosis- central grunting- expiratory seesaw respirations asymmetry
Therapeutic Management of RDS Surfactant replacement therapy Installed into the infant’s trachea Improvement in breathing occurs in minutes Doses repeated prn Other treatment mechanical ventilation correction of acidosis IV fluids
Post Term Infants Born after 42 weeks Increase risk of meconium aspiration Hypoglycemia Loss of subcutaneous fat Skin –peeling, vernix sparse, lanugo absent, fingernails long Focus on prevention – “due date” Attention to thermoregulation & feeding
Meconium Aspiration Syndrome Occurs most often post term infants, decreased amniotic fluid /cord compression Meconium enters lung – obstruction S & S vary from mild to severe respiratory distress: tachypnea, cyanosis, retractions, nasal flaring, grunting Tx – suction at birth, may need warmed, humidified oxygen, or ventilators
Hyperbilirubinemia Pathologic jaundice – occurs within first 24 hours Bilirubin levels >12 in term or 10-14 preterm May lead to kernicterus – brain damage Most common cause – blood incompatibility of mother and fetus, Rh or ABO – only occurs with mother negative Rh or O blood Treatment focus on prevention, assess coombs, monitor bilirubin levels, most common treatment is phototherapy, blood transfusions
Phototherapy for Hyperbilirubinemia Phototherapy – bilirubin on skin changes into water-soluble excreted in bile & urine “Bili” lights placed inside warmer, need patches over eyes, infant wearing only diaper or fiberoptic phototherapy blanket against skin Side effects of phototherapy: freq, loose, green stools, skin changes Can use at home
Other interventions for hyperbilirubinemia Exchange transfusions – if lights not working Maintain neutral thermal environment – not too hot or too cold Provide optimal nutrition – hydrate Protecting the eyes from retinal damage Enhance therapy by expose as much skin as possible to light, remove all clothing except diaper, turn frequently
Infant of a Diabetic Mother Macrosomia – face round, red, body obese, poor muscle tone, irritable, tremors High risk for – trauma during birth, congenital anomalies, RDS, hypocalcemia Hypoglycemia occurs 15-50% of time <40-45 mg/dl, test right after birth, q 2hX4, then q 4 hrX6 until stable Most frequent symptom: jitteriness or tremors Tx – fed, gavage or IV if needed
Hypoglycemia Serum glucose is below 40 mg/dL Tx: feed infant formula or breast milk and retest until glucose stable S & S: jitteriness, lethargy, poor feeding, high-pitched cry, irregular respirations, cyanosis, seizures Risk factors: DM, PIH, preterm, post term, LGA, cold stress, maternal intake of ritodrine or terbutaline
Large for Gestational Age Infants weight is in the 90th % for neonates same gestational age, may be pre, post, or full term infants LGA does not mean post term Infant at risk: birth injuries, hypoglycemia, and polycythemia - macrosomia
Small for Gestational Age Infant whose wt is at or below the 10th % Results from failure to thrive Is a high risk condition SGA does not mean “premature.” Causes: anything restricting uteroplacental blood flow, smoking, DM, PIH, infections Complications: hypoglycemia, meconium aspiration, hypothermia, polycythemia
Mother with Substance Abuse Use of alcohol or illicit drugs Tobacco and alcohol are most frequent Prenatal alcohol exposure is the most commons preventable cause of mental retardation Signs of maternal addition: wt loss, mood swings, constricted pupils, poor hygiene, anorexia, no prenatal care
Drug Withdrawal in Infants Signs of drug exposure opiates – 48-72 hours cocaine – 2-3 days alcohol – within 3-12 hours Symptoms: irritable, hyperactive muscle tone, high-pitched cry High risk for SGA, preterm, RDS, jaundice
Nursing Care of Drug-Exposed Infant Feeding – more difficult may need to gavage Rest – keep stimulation to minimum, reduce noise and lights, calm, slow approach Promote bonding Teach measures for frantic crying: rock, coo, dark room, avoid stimulation
Phenylketonuria - PKU Genetic disorder causes CNS damage from toxic levels of amino acid phenylalanine caused by deficiency of the enzyme phenylalanine hydroxylase Signs- digestive problems, vomiting, seizures, musty odor to urine, mental retardation Tx – low phenylalanine diet – start within 2 months Screening before 24-48 hours needs to be repeated for accuracy
Signs Bonding Delayed Using negative terms describing infant Discussing infant in impersonal terms Failing to give name – check culture Visiting or calling infrequently Decreasing length of visit Refusing to hold infant Lack of eye contact with infant
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