Teri tanosil rasalliklar


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  • maruza
  • 4 kurs tibbiy profilaktika fakulteti
  • Piodermiya, qo’tir, bitlash
  • Piodermiyalar-terini yiringli kasalliklari –stafilokokk, streptokokk, boshqa quzgatuvchilar orqali kelib chiqadigan yiringli kasalliklar guruhi –
  • Uza
  • Chuqur
  • I. Stafilodermiyalar
  • Osteofollikulit
  • Uza follikulit
  • Oddiy sikoz
  • Oddiy xusnbuzar
  • Gudaklarni epidemik pursildoq yarasi
  • Chuqur follikulit
  • Furunkul, furunkulez
  • karbunkul
  • gidradenit
  • II. streptodermiyalar
  • . Streptokokkli impetigo
  • А) Streptokokkli bichilish
  • Б) intertriginoz
  • В) bullez
  • Г) xalqasimon
  • Д) zahmga uxshash
  • Г) yuza panariciy
  • 2. Quruq streptodermiya
  • А)yuzdagi oddiy temiratki
  • 1. Cellulit
  • А) utkir streptoqoqqli
  • 2. Vulgar ektima
  • III. Strepto-stafilodermiya
  • 1. Impetigo oddiy (aralash)
  • Susunkali kechuvchi yarali piodermiya
  • Shankriform piodermiya

Follikulit – soch follikulasini yiringli yalliglanishi Ajratishadi ostiofollikulit, yuza va chuqur follikulit

  • Follikulit – soch follikulasini yiringli yalliglanishi Ajratishadi ostiofollikulit, yuza va chuqur follikulit
  • Oddiy sikoz ko’pincha erkaklarda uchraydigan kasallik
  • Bu jarayenda muylov , soqol va boshqa zonadagi sochlar shikastlanadi

Furunkul – chuqur stafilodermiya.Bu kasallikda soch follikulasini yiringli nekrotik shikastlanish kuzatiladi.Jarayon granulaciya to’qimalari hisobiga chegaralanadi

  • Furunkul – chuqur stafilodermiya.Bu kasallikda soch follikulasini yiringli nekrotik shikastlanish kuzatiladi.Jarayon granulaciya to’qimalari hisobiga chegaralanadi
  • Karbunkul -bir nechta soch follikullarning yiringli nekrotik yalliglanishi . Yalliglanuvchi infiltrat paydo bo’ladi, derma , gipodermada joylashadi,kattaligi bolalar kaftiday bo’lishi mumkin Bir nechta yiringli nekrotik yalliglanuvchi tugunlar bo’ladi

Gidradenit apokrin bezlarni yiringli yalliglanishi. Stafilokokklar bezlarning yuliga teridagi travmalar orqali ( kupincha soch olinganda kiradin.

  • Gidradenit apokrin bezlarni yiringli yalliglanishi. Stafilokokklar bezlarning yuliga teridagi travmalar orqali ( kupincha soch olinganda kiradin.

Chaqaloqlarning epidemik pursildoq yarasi Kupincha bola tugilganda 3-5 kundan keyin paydo buladi - kontagioz yuza stafilodermiya

  • Chaqaloqlarning epidemik pursildoq yarasi Kupincha bola tugilganda 3-5 kundan keyin paydo buladi - kontagioz yuza stafilodermiya

Impetigo – kontagioz kasallik . Nofollikular , qopqogi ilvillagan , atrofida qizil gardishi ,ishida seroz yiringli suyuqligi bor Pufaklar paydo buladi

  • Impetigo – kontagioz kasallik . Nofollikular , qopqogi ilvillagan , atrofida qizil gardishi ,ishida seroz yiringli suyuqligi bor Pufaklar paydo buladi

Ektima – terini chuqur streptokokkli shiqastlanishi. Bu chuqur yara chetlari kutarilib turadi, tubi yumshoq yiringli , atrofida ogriqli yalliglanuvchi infiltrat kuzatiladi

  • Ektima – terini chuqur streptokokkli shiqastlanishi. Bu chuqur yara chetlari kutarilib turadi, tubi yumshoq yiringli , atrofida ogriqli yalliglanuvchi infiltrat kuzatiladi

Surunkali kechuvchi vegetaciyali piodermiya - chuqur piodermiyaning surunkali kechuvchi aralash strepto-stafilokokkli turi .

  • Surunkali kechuvchi vegetaciyali piodermiya - chuqur piodermiyaning surunkali kechuvchi aralash strepto-stafilokokkli turi .
  • Shankriform piodermiya – surunkali kechuvchi yarali piodermiya . Zaxmdagi shankrni eslatadi

Qutir

  • qutir - teri parazitar kasalligi quzgatuvchisi - qutir kanasi Sarcoptes scabiei hominis( odamning teri ichida joylashadi )

Yuqish yullari :

  • Yuqish yullari :
  • Kontakt - maichiy yuli
  • jinsiy yo’l

Klinik manzara

  • Qichishish
  • Qo’tir yullari
  • Klinik alomatlarini harakterli joylashishi
  • Qichishish kechqurun eki kechasi kupayadi

qutir tashxisini quyidagiga asoslanadi :

  • qutir tashxisini quyidagiga asoslanadi :
  • KLINIK MANZARA
  • EPIDEMIOLOGIK MA’LUMOTLAR
  • Laboratoriya usullarii

Qiesiy tashhis

  • Hayvonlarda uchraydigan qutir
  • Neyrodermit
  • Qichima
  • Davolash
  • benzilbenzoat suv sovunli emulsiya
  • Demyanovich usuli №1 – 60% eritma natriy tiosulfat va №2 – 6% р-р HCL .
  • 20-33% oltin gugurt malhami .

Bitlash

  • Bitlash
  • Bosh pedikulezi
  • Kiyim pedikulezi
  • Chov pedikulezi(ftiriaz)

Bosh pedikulezi

  • Bitlar qon so’rganda teri ichra ularning sulagi qoladi . Sulakning tasiri natijasida tishlagan joyida qishidigan zich tugunchalar paydo bo’ladi .
  • Bosh biti rikketsiozlar tarqalishiga sababchi bo’ladi (toshma ,qaytarma tifi va xok ) Qattiq qichishish piodermiya rivojlanishiga olib kelishi mumkin

bosh bitni davolash

  • 10% suvli sovunli kerosinli emulsiya
  • 0,5% eritva permetrin (nittifor eki Loncid )
  • Benzilbenzoat malhami
  • Shampun «anti-bit »
  • Losyon «Valiten »
  • 0,15% karbofosni suvli emulsiyasi (bitlarni hamma bosqichida uldiradi )

Kiyim bitini davolash

  • Kiyim bitini davolash
  • Sovun bilan chumilish , kiyim almashtirish,desincekciya qilish .
  • Qichiganda terini kuniga 2-3 mahal 4% karbol eki 1% mentol spirti bilan 7-10 kun ichida artadi

davolash

  • 0,15% karbofosni suvli emulsiyasi
  • 20% benzilbenzoatni suvli emulsiyasi
  • 5% degtyar eki oltin gugurt malhami.
  • Chov sohasida sochlarni olib tashlab sovun bilan chumilish
  • The subject: “«PYODERMIA; Scabies, pediculosis»
  • Pyoderma, a large group of dermatosis caused festering cocci, mainly staphylococcus and streptococci. Their could association with other microorganisms (Proteus vulgar, mycoplasma, etc.). There pyodermia as a primary skin disease or other complications dermatosis (itchy or allergic dermatosis) as a result of mikrotraumas, with skin from the outside or the activation of pathogenic strains of microbial autoflory
  • The main conditions for the development of pyodermia is contact with a pathogen, the presence of the gate and an enabling environment for the introduction of the causative agent, endogenous factors, hereditary or acquired immunodeficiency, reducing antibacterial protection body, the activation of microbial flora (the factor of «aggression» and «protection», long receiving corticosteroids, cytostatics, immunosuppressant, leather shift RN in alkaline side). Transmission, airborne, mechanical pernos piococci from the center of lesions, through semination clothes, objects. According to etiological principle distinguish piodermity: stafilodermii (ostiofollikulit, folliculitis, furuncle, carbuncle, and hydradenitis) streptogenes impetigo, ekthima, mixed pyodermia.
  • Treatment-antibiotics, immunotherapy, vitamin therapy, local therapy
  • Scabies
  • Ways of contracting scabies: - contact, consumer (direct and indirect); sexual contact. Characteristic space localization itch rash: - intermanual folds brushes - flexion of the upper and lower limbs - the body: in the waist, abdomen, anterior surface podkryltsovyh cavities; - in areole breasts in women - in the area of the penis men. The basic have clinical signs of scabies are: - increasing itching at night, a characteristic rash localization, the presence of paired papules, papule, vesicle, traces itches secondary pustules; symptom Gorchakova –Ardi.
  • Treatment of specific ant scratches therapy
  • Pediculosis.
  • Parasitizing human lice on the skin, accompanied by changes in the skin, as a result of bites. There are 3 types of lice parasitic on human skin: - headaches - pubic (ftiriaz) - carpotis. The main clinical signs of lice: - finding lice - Finding nits - itching skin; - Excoriation, bites in the form of hemorrhagic spots, blisters, secondary piodermitis elements.    Treatment: Specific treatment against pediculosis
  • The subject: “«PYODERMIA; Scabies, pediculosis»
  • Pyoderma, a large group of dermatosis caused festering cocci, mainly staphylococcus and streptococci. Their could association with other microorganisms (Proteus vulgar, mycoplasma, etc.). There pyodermia as a primary skin disease or other complications dermatosis (itchy or allergic dermatosis) as a result of mikrotraumas, with skin from the outside or the activation of pathogenic strains of microbial autoflory
  • The main conditions for the development of pyodermia is contact with a pathogen, the presence of the gate and an enabling environment for the introduction of the causative agent, endogenous factors, hereditary or acquired immunodeficiency, reducing antibacterial protection body, the activation of microbial flora (the factor of «aggression» and «protection», long receiving corticosteroids, cytostatics, immunosuppressant, leather shift RN in alkaline side). Transmission, airborne, mechanical pernos piococci from the center of lesions, through semination clothes, objects. According to etiological principle distinguish piodermity: stafilodermii (ostiofollikulit, folliculitis, furuncle, carbuncle, and hydradenitis) streptogenes impetigo, ekthima, mixed pyodermia.
  • Treatment-antibiotics, immunotherapy, vitamin therapy, local therapy
  • Scabies
  • Ways of contracting scabies: - contact, consumer (direct and indirect); sexual contact. Characteristic space localization itch rash: - intermanual folds brushes - flexion of the upper and lower limbs - the body: in the waist, abdomen, anterior surface podkryltsovyh cavities; - in areole breasts in women - in the area of the penis men. The basic have clinical signs of scabies are: - increasing itching at night, a characteristic rash localization, the presence of paired papules, papule, vesicle, traces itches secondary pustules; symptom Gorchakova –Ardi.
  • Treatment of specific ant scratches therapy
  • Pediculosis.
  • Parasitizing human lice on the skin, accompanied by changes in the skin, as a result of bites. There are 3 types of lice parasitic on human skin: - headaches - pubic (ftiriaz) - carpotis. The main clinical signs of lice: - finding lice - Finding nits - itching skin; - Excoriation, bites in the form of hemorrhagic spots, blisters, secondary piodermitis elements.    Treatment: Specific treatment against pediculosis



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